Endothelial HIF-2α contributes to severe pulmonary hypertension due to endothelial-to-mesenchymal transition Article

Tang, Haiyang, Babicheva, Aleksandra, McDermott, Kimberly M et al. (2018). Endothelial HIF-2α contributes to severe pulmonary hypertension due to endothelial-to-mesenchymal transition . American Journal of Physiology - Lung Cellular and Molecular Physiology, 314(2), L256-L275. 10.1152/ajplung.00096.2017

Open Access International Collaboration

cited authors

  • Tang, Haiyang; Babicheva, Aleksandra; McDermott, Kimberly M; Gu, Yali; Ayon, Ramon J; Song, Shanshan; Wang, Ziyi; Gupta, Akash; Zhou, Tong; Sun, Xutong; Dash, Swetaleena; Wang, Zilu; Balistrieri, Angela; Zheng, Qiuyu; Cordery, Arlette G; Desai, Ankit A; Rischard, Franz; Khalpey, Zain; Wang, Jian; Black, Stephen M; Garcia, Joe GN; Makino, Ayako; Yuan, Jason X-J

sustainable development goals

authors

publication date

  • February 1, 2018

published in

keywords

  • ANIMAL-MODELS
  • ARTERIAL-HYPERTENSION
  • GROWTH-FACTOR-BETA
  • HIF-ALPHA
  • HYDROXYLASE DOMAIN PROTEIN-2
  • HYPOXIA-INDUCIBLE FACTORS
  • Life Sciences & Biomedicine
  • MICE
  • OXYGEN
  • PROMOTES
  • Physiology
  • Respiratory System
  • SMOOTH-MUSCLE-CELLS
  • Science & Technology
  • endothelial cell
  • intimal lesion
  • prolyl hydroxylase domain-containing protein
  • pulmonary arterial hypertension

Digital Object Identifier (DOI)

start page

  • L256

end page

  • L275

volume

  • 314

issue

  • 2