Corrigendum to “Didymin induces apoptosis through mitochondrial dysfunction and up-regulation of RKIP in human hepatoma cells” [Chem. Biol. Interact. 261 (2017) 118-126] (Chemico-Biological Interactions (2017) 261(118-126) (S0009279716306470), (10.1016/j.cbi.2016.11.026))
Other Scholarly Work
Wei, J, Huang, Q, Bai, F et al. (2021). Corrigendum to “Didymin induces apoptosis through mitochondrial dysfunction and up-regulation of RKIP in human hepatoma cells” [Chem. Biol. Interact. 261 (2017) 118-126] (Chemico-Biological Interactions (2017) 261(118-126) (S0009279716306470), (10.1016/j.cbi.2016.11.026))
. 334 10.1016/j.cbi.2020.109338
Wei, J, Huang, Q, Bai, F et al. (2021). Corrigendum to “Didymin induces apoptosis through mitochondrial dysfunction and up-regulation of RKIP in human hepatoma cells” [Chem. Biol. Interact. 261 (2017) 118-126] (Chemico-Biological Interactions (2017) 261(118-126) (S0009279716306470), (10.1016/j.cbi.2016.11.026))
. 334 10.1016/j.cbi.2020.109338
The authors regret that the wrong Figs. 3, 6B, 8 and 9 were published. The correct figures are provided in this corrigendum. The authors would like to apologise for any inconvenience caused. [Figure presented] Fig. 3. OVD inhibited HepG2 cell clonogenicity and migratory potential. (A) Clonogenicity assay showed that OVD suppressed HepG2 cell colony formation. (B) OVD treatment resulted in a significant decrease in migration ability. (C) OVD treatment decreased the ratio of migration. [Figure presented] Fig. 6. OVD induced loss of MMP and mitochondrial cytochrome c release in HepG2 cells. (B) Cytochrome c expression was determined by Western blot analysis. The numbers 1 to 4 represented the control cells group, OVD group, locostatin. Group, and OVD + locostatin group, respectively. [Figure presented] Fig. 8. OVD enhanced RKIP expression, but inhibited the ERK/MAPK pathway in HepG2 cells. (A to E) The expression of RKIP, MEK, p-MEK, ERK, p-ERK, p38, p-p38, JNK and p-JNK were detected by western blot, respectively. The bands 1 to 4 represented the control cells group, OVD group, locostatin group, and OVD + locostatin group, respectively. *P < 0.05 vs. control cells group and ▲P < 0.05 vs. OVD + locostatin group. (F) siRNA assay was used to silence RKIP expression. The bands I to V represented the control cells group, OVD group, Control-siRNA group, RKIP-siRNA group, and OVD + RKIP-siRNA group, respectively. *P < 0.05 vs. control cells group, ▲P < 0.05 vs. OVD + RKIP-siRNA group, and △P < 0.05 vs. OVD + Control-siRNA group. [Figure presented] Fig. 9. OVD inhibited PI3K/Akt pathway in HepG2 cells. PI3K, Akt and p-Akt expression was detected by western blot. The bands 1 to 4 represented the control cells group, OVD group, locostatin group, and OVD + locostatin group, respectively. Data were expressed as the means ± SD. *P < 0.05 vs. control cells group and ▲P < 0.05 vs. OVD + locostatin group.
