The neurotoxicity of amyloid β-protein oligomers is reversible in a primary neuron model Article

Tanokashira, Daisuke, Mamada, Naomi, Yamamoto, Fumiko et al. (2017). The neurotoxicity of amyloid β-protein oligomers is reversible in a primary neuron model . MOLECULAR BRAIN, 10 10.1186/s13041-016-0284-5

Open Access International Collaboration

cited authors

  • Tanokashira, Daisuke; Mamada, Naomi; Yamamoto, Fumiko; Taniguchi, Kaori; Tamaoka, Akira; Lakshmana, Madepalli K; Araki, Wataru

sustainable development goals

authors

publication date

  • January 31, 2017

published in

keywords

  • A-BETA
  • ACTIVATION
  • ALZHEIMERS-DISEASE
  • Alzheimer's disease
  • Amyloid beta-protein
  • CATENIN
  • CULTURED CORTICAL-NEURONS
  • GLYCOGEN-SYNTHASE KINASE-3-BETA
  • HIPPOCAMPAL-NEURONS
  • HYPERPHOSPHORYLATION
  • Life Sciences & Biomedicine
  • NEURODEGENERATIVE DISEASES
  • Neurosciences
  • Neurosciences & Neurology
  • Neurotoxicity
  • Oligomer
  • Primary neuron
  • Science & Technology
  • TAU PHOSPHORYLATION
  • Tau
  • beta-catenin

Digital Object Identifier (DOI)

publisher

  • BMC

volume

  • 10