p21(ras) Activation following hypoxia-ischemia in the newborn rat brain is dependent on nitric oxide synthase activity but p21(ras) does not contribute to neurologic injury Article

Wainwright, MS, Brennan, LA, Dizon, ML et al. (2003). p21(ras) Activation following hypoxia-ischemia in the newborn rat brain is dependent on nitric oxide synthase activity but p21(ras) does not contribute to neurologic injury . 146(1-2), 79-85. 10.1016/j.devbrainres.2003.09.012

keywords

  • ADENOSINE
  • CALCIUM
  • DAMAGE
  • Developmental Biology
  • INHIBITION
  • Life Sciences & Biomedicine
  • MICE
  • NEONATAL-RAT
  • NEUROPROTECTION
  • Neurosciences
  • Neurosciences & Neurology
  • POTENTIATION
  • PROTEIN TRANSFERASE
  • Ras
  • SUPPRESSION
  • Science & Technology
  • brain
  • disorders of the nervous system
  • hypoxia-ischemia
  • ischemia
  • neonate
  • nitric oxide synthase

Digital Object Identifier (DOI)

publisher

  • ELSEVIER SCIENCE BV

start page

  • 79

end page

  • 85

volume

  • 146

issue

  • 1-2