Ginsenoside Rg3 antagonizes adriamycin-induced cardiotoxicity by improving endothelial dysfunction from oxidative stress via upregulating the Nrf2-ARE pathway through the activation of akt Article

Wang, Xiaoying, Chen, Lili, Wang, Ting et al. (2015). Ginsenoside Rg3 antagonizes adriamycin-induced cardiotoxicity by improving endothelial dysfunction from oxidative stress via upregulating the Nrf2-ARE pathway through the activation of akt . PHYTOMEDICINE, 22(10), 875-884. 10.1016/j.phymed.2015.06.010

cited authors

  • Wang, Xiaoying; Chen, Lili; Wang, Ting; Jiang, Xiaoqing; Zhang, Han; Li, Pan; Lv, Bin; Gao, Xiumei

sustainable development goals

authors

publication date

  • September 15, 2015

published in

keywords

  • ACUTE DOXORUBICIN CARDIOTOXICITY
  • Adriamycin
  • Aortic ring
  • CARDIAC INJURY
  • CELLS
  • Cardiotoxicity
  • Chemistry, Medicinal
  • EXPRESSION
  • GROWTH
  • Ginsenoside Rg3
  • INDUCED APOPTOSIS
  • Integrative & Complementary Medicine
  • Life Sciences & Biomedicine
  • Nrf2
  • Oxidative stress
  • PROTECTS
  • Pharmacology & Pharmacy
  • Plant Sciences
  • REACTIVE OXYGEN
  • SUPPRESSION
  • Science & Technology
  • TRANSCRIPTION FACTOR

Digital Object Identifier (DOI)

publisher

  • ELSEVIER GMBH

start page

  • 875

end page

  • 884

volume

  • 22

issue

  • 10