High Ca2+ Influx During Traumatic Brain Injury Leads to Caspase-1-Dependent Neuroinflammation and Cell Death Article

Abdul-Muneer, PM, Long, Mathew, Conte, Adriano Andrea et al. (2017). High Ca2+ Influx During Traumatic Brain Injury Leads to Caspase-1-Dependent Neuroinflammation and Cell Death . MOLECULAR NEUROBIOLOGY, 54(6), 3964-3975. 10.1007/s12035-016-9949-4

Open Access

cited authors

  • Abdul-Muneer, PM; Long, Mathew; Conte, Adriano Andrea; Santhakumar, Vijayalakshmi; Pfister, Bryan J

sustainable development goals

authors

publication date

  • August 1, 2017

published in

keywords

  • APOPTOSIS
  • Apoptosis
  • CEREBROSPINAL-FLUID
  • Ca2+ influx
  • Caspase-1
  • Fluid percussion injury
  • IL-1 beta
  • Life Sciences & Biomedicine
  • MATRIX METALLOPROTEINASES
  • MECHANISMS
  • MITOCHONDRIAL PERMEABILITY TRANSITION
  • MODEL
  • NLRP3 INFLAMMASOME
  • Neuroinflammation
  • Neuronal stretch injury
  • Neurosciences
  • Neurosciences & Neurology
  • OXIDATIVE STRESS
  • RECEPTOR
  • Science & Technology
  • TETRODOTOXIN
  • Traumatic brain injury

Digital Object Identifier (DOI)

publisher

  • SPRINGER

start page

  • 3964

end page

  • 3975

volume

  • 54

issue

  • 6