ICAM-1 Deletion Using CRISPR/Cas9 Protects the Brain from Traumatic Brain Injury-Induced Inflammatory Leukocyte Adhesion and Transmigration Cascades by Attenuating the Paxillin/FAK-Dependent Rho GTPase Pathway Article

Saikia, Bibhuti Ballav, Bhowmick, Saurav, Malat, Anitha et al. (2024). ICAM-1 Deletion Using CRISPR/Cas9 Protects the Brain from Traumatic Brain Injury-Induced Inflammatory Leukocyte Adhesion and Transmigration Cascades by Attenuating the Paxillin/FAK-Dependent Rho GTPase Pathway . JOURNAL OF NEUROSCIENCE, 44(11), 10.1523/JNEUROSCI.1742-23.2024

Open Access

cited authors

  • Saikia, Bibhuti Ballav; Bhowmick, Saurav; Malat, Anitha; Rani, MR Preetha; Thaha, Almas; Abdul-Muneer, PM

sustainable development goals

authors

publication date

  • March 13, 2024

published in

keywords

  • CRISPR/Cas9
  • DAMAGE
  • DYSFUNCTION
  • GROWTH
  • LFA-1
  • Life Sciences & Biomedicine
  • MAC-1
  • MATRIX
  • MOLECULE-1
  • NEUROINFLAMMATION
  • Neurosciences
  • Neurosciences & Neurology
  • RECOVERY
  • Rho GTPase
  • Science & Technology
  • TYROSINE PHOSPHORYLATION
  • functional recovery
  • intercellular adhesion molecule-1
  • leukocytes
  • paxillin - FAK
  • transmigration
  • traumatic brain injury

Digital Object Identifier (DOI)

publisher

  • SOC NEUROSCIENCE

volume

  • 44

issue

  • 11