Sab mediates mitochondrial dysfunction involved in imatinib mesylate-induced cardiotoxicity Article

Chambers, Tara P, Santiesteban, Luis, Gomez, David et al. (2017). Sab mediates mitochondrial dysfunction involved in imatinib mesylate-induced cardiotoxicity . TOXICOLOGY, 382 24-35. 10.1016/j.tox.2017.03.006

cited authors

  • Chambers, Tara P; Santiesteban, Luis; Gomez, David; Chambers, Jeremy W

sustainable development goals

authors

publication date

  • May 1, 2017

published in

keywords

  • C-Jun N-terminal Kinase (JNK)
  • CHRONIC MYELOID-LEUKEMIA
  • Cardiomyocyte
  • Cell death
  • FAILURE
  • GASTROINTESTINAL STROMAL TUMORS
  • INHIBITION
  • Imatinib
  • JNK TRANSLOCATION
  • Life Sciences & Biomedicine
  • Mitochondria
  • N-TERMINAL KINASE
  • PHASE-III
  • Pharmacology & Pharmacy
  • Sab
  • Science & Technology
  • THERAPEUTIC AGENT
  • TOXICITY
  • TYROSINE KINASE
  • Toxicology

Digital Object Identifier (DOI)

publisher

  • ELSEVIER IRELAND LTD

start page

  • 24

end page

  • 35

volume

  • 382